Absolutely, says Katherine Hamilton, MD , an assistant professor of clinical neurology and a headache specialist at Penn Medicine in Philadelphia. Research suggests that the association between migraine and sleep is bidirectional.
Headache can cause disturbances in sleep, and sleep problems can trigger a migraine attack, according to a review published in January in Therapeutic Advances in Neurological Disorders. The good news is that good sleep habits are one of the most effective tools for managing migraine, says Lauren Doyle Strauss, DO , a headache specialist and assistant professor at Wake Forest Baptist Health in Winston-Salem, North Carolina.
In some individuals, improving sleep hygiene can lead to improvements in migraine in just a few months or less, according to the American Headache Society. Not getting enough sleep and sleeping too much can both be headache triggers, according to the American Migraine Foundation. The organization recommends getting between seven and eight hours of sleep each night.
When possible, try to go to bed and get up at about the same time each day, suggests Hamilton. Avoiding naps can help you sleep better at night, says Dr. Bedtime rituals such as taking a warm bath, reading, listening to calming music, doing yoga, or praying can help you fall asleep, according to the American Migraine Foundation. Rosen recommends a cool, dark, and quiet room for sleeping. Keep your bedroom uncluttered , and try blackout curtains if you are light sensitive, the National Sleep Foundation suggests.
He recommends shutting down all electronics an hour prior to bedtime. Rosen recommends paying attention to how your diet and fluid intake affect your sleep. Cortical spreading depression triggers migraine attack.
Intrinsic brain activity triggers trigeminal meningeal afferents in a migraine model. Nat Med. Supression of cortical spreading depression in migraine prophylaxis. Review paper discussing the several phases of migraine attacks in the light of neurophysiological changes. Kelman L. The premonitory symptoms prodrome : a tertiary care study of migraineurs. Hypothalamic activation in spontaneous migraine attacks. PubMed Google Scholar. Distraction modulates connectivity of the cingulo-frontal cortex and the midbrain during pain—an fMRI analysis.
Diencephalic and brainstem mechanisms in migraine. Nat Rev Neuroscience. Pivotal paper focusing on the role of subcortical structures in migraine.
It highlights the role of the hypothalamus and of the orexin peptides in the modulation of pain. Brain stem activation in spontaneous human migraine attacks. A positron emission tomographic study in spontaneous migraine.
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Release of vasoactive peptides in the extracerebral circulation of humans and the cat during activation of the trigeminovascular system. Hargreaves R. New migraine and pain research. Headache ; Suppl 1:S26—S Neurogenically mediated plasma extravasation in dura mater: effect of ergot alkaloids. A possible mechanism of action in vascular headache. Retinal plasma extravasation in animals but not in humans: implications for the pathophysiology of migraine.
Up-regulated neuronal COX-2 expression after cortical spreading depression is involved in non-REM sleep induction in rats. J Neurosci Res. Local energy depletion in the basal forebrain increases sleep. Eur J Neurosci.
Unilateral cortical spreading depression affects sleep need and induces molecular and electrophysiological signs of synaptic potentiation in vivo. Cereb Cortex. Paper correlating the influence that unilateral cortical spreading depression would have on behavior. Clock genes, oscillators and cellular networks in the suprachiasmatic nuclei. J Biol Rhythms. Sleep neurobiology from a clinical perspective.
Hypothalamic regulation of sleep and circadian rhythms. Siegel JM. Hypocretin orexin : role in normal behavior and neuropathology. Annu Rev Psychol. Orexin receptor function, nomencalture and pharmacology. Pharmacol Rev. Sakurai T. The neuronal circuit of orexin hypocretin : maintaining sleep and wakefulness. Nat Rev Neurosci. Hungs M, Mignot E. Orexin receptors as therapeutic targets. Prog Brain Res. Spinal orexin-1 receptors mediate anti-hyperalgesic effects of intrathecally-administered orexins in diabetic neuropathic pain model rats.
Brain Res. Enhanced nociception by intraceebroventricularly and intrathecally adminsitered orexin A and B hypocretin 1 and 2 in mice. Orexin-A an hypothalamic peptide with anlgesic properties. Common sleep disorders include: insomnia, sleep apnea, teeth grinding.
Headaches that are linked to sleep include: wake-up headache and hypnic headache. Common Sleep Disorders Insomnia The most common sleep problem for people living with migraine is insomnia. Snoring and Sleep Apnea Regular snoring is a risk factor for chronic daily headache. Teeth Grinding Waking up with a dull, constant headache or sore jaw can be a symptom of excessive teeth grinding, which can contribute to temporomandibular disorders.
Types of Headaches Linked to Sleep Wake-up Headache The most common time for migraine to occur is in the early morning. Hypnic Headache Hypnic headache is a rare, primary headache disorder characterized by frequently occurring headaches that happen only during sleep. Cluster Headache Cluster headaches are another primary headache that may develop during sleep. The Link Between Sleep, Headache and Mood The same brain regions and chemical messengers impact sleep, headache and mood, so inadequate or poor quality sleep increases the odds for headache and mood change.
The Importance of Healthy Sleep Behavioral sleep changes can promote restful, regular sleep and reduce headache. Font Size. Seasonal Migraine Triggers. Managing Diet and Migraine. Nahas, MD. Why Do I Have Migraine? What Causes Migraine. Living With Migraine: Diet and Migraine. Join Our Newsletter.
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